Effect of chitosan on anthracnose disease and physiology of harvested chili ‘Jinda’

บทความในวารสาร

ผู้เขียน/บรรณาธิการ

กลุ่มสาขาการวิจัยเชิงกลยุทธ์

ไม่พบข้อมูลที่เกี่ยวข้อง

รายละเอียดสำหรับงานพิมพ์

รายชื่อผู้แต่ง: Jitareerat P., Uthairatanakij A., Aiamla-or S.

ผู้เผยแพร่: International Society for Horticultural Science (ISHS)

ปีที่เผยแพร่ (ค.ศ.): 2017

วารสาร: Acta Horticulturae (0567-7572)

Volume number: 1179

หน้าแรก: 119

หน้าสุดท้าย: 124

จำนวนหน้า: 6

ISBN: 9789462611757

นอก: 0567-7572

eISSN: 2406-6168

URL: https://www.scopus.com/inward/record.uri?eid=2-s2.0-85037683428&doi=10.17660%2fActaHortic.2017.1179.18&partnerID=40&md5=c2249bf91754f51554befa64165be26d

ภาษา: English-Great Britain (EN-GB)

ดูบนเว็บไซต์ของสำนักพิมพ์

บทคัดย่อ

Chili pepper fruits ‘Jinda’ were wound-inoculated with the anthracnose pathogen Colletotrichum gloeosporioides, and then coated with chitosan solution at 0 (control), 1.2 or 1.6% (w/v). Chitosan delayed disease development and weight loss, with 1.6% giving better results than 1.2%. However, 1.6% chitosan caused fruit fermentation. Chitosan coating at 1.2% reduced respiration rate and ethylene production, but did not delay firmness loss or color changes (L* and a* values). The effect of 1.2% chitosan coating on the induction of peroxidase (POD), phenylalanine-ammonia lyase (PAL), β-1,3-glucanase (GLU) and chitinase (CHI) activities was determined in chili fruits after wound-inoculation with C. gloeosporioides for 0 to 48 h. The result showed that chitosan coating had not induced these enzymes, but POD, GLU and CHI increased in response to wounding. This result implies that the delayed disease development may not be caused by an indirect effect of chitosan to induce plant defense response, but by its direct effect.

คำสำคัญ

3-glucanase, Beta-1, Capsicum sp., Phenylalanine-ammonia lyase